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Our data identify G6PD modulation as a novel mechanism exploited by influenza virus to induce oxidative stress in host cells and favor its replication and suggest that the rescue of antioxidant response could represent a new cell-targeted strategy to interfere with influenza virus infection

Because the stratum corneum is lipid-rich, peptides do not penetrate easily

This release of secondary messenger molecules, such as IP3, might potentially stimulate the discharge of calcium ions (Ca2+) from the cells internal stores

Single-compound intervention arms with saline controls are the most common design in the published literature

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We observed that although BET effectively lowered homocysteine levels, it did not reduce N 2 O-induced CPP, suggesting that reducing homocysteine alone is insufficient to attenuate reward-related behavior