FRP-2 Polyclonal Antibody, 50ul Microbial Genome Editing Defects in ATG16L1 are a

Defects in ATG16L1 are a cause of susceptibility to inflammatory bowel disease type 10 (IBD10)

and it may regulate the migration and recruitment of dentritic and T cells during inflammatory and immunological responses

A similar protein in rodents is thought to play a role in in the regulation of neuronal signaling pathways

DLAB's new generation of fully autoclavable manual pipettes feature an excellent ergonomic design with an ultra-light body weighing less than 100g

Stimulates the autophosphorylation and kinase activity of STK38 and STK38L

FRP-2 Polyclonal Antibody, 50ul Microbial Genome Editing Defects in ATG16L1 are aSFRP2 encodes a member of the SFRP family that contains a cysteine rich domain homologous to the putative Wnt binding site of Frizzled proteins. SFRPs act as soluble modulators of Wnt signaling. Methylation of SFRP2 is a potential marker for the presence of colorectal cancer.