AGT2 Polyclonal Antibody, 20ul Cell Separation and Collection Defects in MEF2A could be

Defects in MEF2A could be a cause of autosomal dominant coronary artery disease 1 with myocardial infarction (ADCAD1)

and phopholipase D activation

CCAAT/enhancer binding protein beta functions as a homodimer but can also form heterodimers with CCAAT/enhancer-binding proteins alpha

It encodes a type 1 membrane bound protease that is expressed in many tissues

PBC enventually leads to cirrhosis and liver failure

AGT2 Polyclonal Antibody, 20ul Cell Separation and Collection Defects in MEF2A could beThe protein encoded by this gene is a class III pyridoxal phosphate dependent mitochondrial aminotransferase. It catalyzes the conversion of glyoxylate to glycine using L alanine as the amino donor. It is an important regulator of methylarginines and is involved in the control of blood pressure in kidney. Polymorphisms in this gene affect methylarginine and beta aminoisobutyrate metabolism, and are associated with carotid atherosclerosis. Alternative