GATA-2/3 Polyclonal Antibody, 20ul Protein Expression Defects in ATG16L1 are a

Defects in ATG16L1 are a cause of susceptibility to inflammatory bowel disease type 10 (IBD10)

which form a tetrameric holoenzyme

including interleukin 6 (IL6)

Mutations in F2 lead to various forms of thrombosis and dysprothrombinemia

One of several members of the Ser/Thr protein kinase family

GATA-2/3 Polyclonal Antibody, 20ul Protein Expression Defects in ATG16L1 are aGATA3 encodes GATA binding protein 3 which belongs to the GATA family of transcription factors. The protein contains two GATA type zinc fingers and is an important regulator of T cell development and plays an important role in endothelial cell biology. Defects in this gene are the cause of hypoparathyroidism with sensorineural deafness and renal dysplasia.