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The peptide has been characterized in tendon, ligament, muscle, gut, vascular, neural and bone injury models, where it consistently accelerates structural recovery, restores barrier function and rescues vascular perfusion through a coordinated upregulation of growth factor receptors, the nitric oxide system and angiogenic signaling [13]

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17 These findings again highlighted the key function of specific TJ, integral or associated, proteins in regulating barrier function and epithelial homeostasis, which together could help explain altered expression of these proteins in IBD patients